There’s a growing number of severe acute hepatitis cases of unknown etiology in kids that mysteriously keep popping up. The problem is, at least in the USA, we don’t historically see this specific type of hepatitis clustered in such a short period of time. So in this video we’ll go over what hepatitis actually is, what it’s usually caused by, and then we’ll explore the data on what could be causing these new severe hepatitis cases in children. That said, we’ll go through my some of my newest Substack posts that include data points, and images which will help simplify all of this for you.
REFERENCES:
https://www.journal-of-hepatology.eu/action/showPdf?pii=S0168-8278%2822%2900234-3
https://www.who.int/health-topics/hepatitis#tab=tab_1
LETS CONNECT:
It's good that you made a distinction between association/correlation and causation. (I think of it it as, "Correlation doesn't equal causation.")
As a side note, I think that's going to be a hugely significant talking point, not just for this particular topic, but many other health-related ones as well.
Anyway, it's certainly unfourtunate that this is happening to children.
Every avenue should be looked at. Here are just a few things that come to my mind:
1. We have to consider that there may not just be one absolute cause. Instead, there may be a number of contributing factors.
I have a feeling that, in order to explore this, we may have to challenge some of the things we knew in the past.
That is, we have to be willing to say that because variables 1, 2, and 3 led to hepatitis in the past, that doesn't necessarily hold true for these new cases.
There could be a new, or previously-unknown explanation.
We have to look at something old with new eyes.
2. That technical paper said "non A-E." Originally, I thought it meant neither type A nor type E, but then I thought about this more: it could mean this hepatitis doesn't fall under the A-E range.
So...could it be a hepatitis type...F?
Is that the right train of thought, or am I completely off on that?
3. Are there any observable variables that distinguish those who recovered vs those who didn't? Or those who needed transplants vs. those who didn't?
4. You said, historically speaking, we don't see these clusters. But, do we have any such cases in our history, anywhere in the world? If so, what can we learn from those cases?
5. Can we observe any genetic predispositions?
6. Were these children immunocompromised? Did they have other illnesses or conditions?
7. Vax status: The age range of the 9 Alabama children was from 0 to 6. I think that currently (at least in the UK), the minimum age for covid vaccination is 5. (I think it's the same for the US, but please double-check, as it may depend on the dates authorization was given.)
I'm not sure how the vaxx stats play into this, because it seems that at least one of these children is too young to have taken the vaxx. (But again, please double-check this.)
8. Do these children live close together, geographically-speaking? (I know this is being observed in a number of countries. I mean within those countries--does geography play a role? Could there be something environmental, or that has to do with the water and food systems that could be contributing to this?)
Those are my thoughts. Unfortunately, time limits how much I can contribute to this discussion, but hopefully, what I've stated above is of some value.